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CHIR99021 and fibroblast growth factor 1 enhance the regenerative potency of human cardiac muscle patch after myocardial infarction in mice.
Cardiomyocytes from CCND2-overexpressing human induced-pluripotent stem cells repopulate the myocardial scar in mice: A 6-month study.
Sam68 impedes the recovery of arterial injury by augmenting inflammatory response.
Cardiomyocyte-GSK-3α promotes mPTP opening and heart failure in mice with chronic pressure overload.
HDAC inhibition induces autophagy and mitochondrial biogenesis to maintain mitochondrial homeostasis during cardiac ischemia/reperfusion injury.
HDAC inhibition as a therapeutic strategy in myocardial ischemia/reperfusion injury.
Genetic deletion of 12/15 lipoxygenase promotes effective resolution of inflammation following myocardial infarction.
The stress kinase JNK regulates gap junction Cx43 gene expression and promotes atrial fibrillation in the aged heart.
Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis.
Genetic disruption of the cardiomyocyte circadian clock differentially influences insulin-mediated processes in the heart.
Regulation of cardiac gap junctions by protein phosphatases.
Myocardial relaxation is accelerated by fast stretch, not reduced afterload.
Cardiac inflammation in genetic dilated cardiomyopathy caused by MYBPC3 mutation.
A computational model of cardiac fibroblast signaling predicts context-dependent drivers of myofibroblast differentiation.
Computational modeling of cardiac fibroblasts and fibrosis.
Modifying the mechanics of healing infarcts: Is better the enemy of good?.
ATP sensitive K+ channels are critical for maintaining myocardial perfusion and high energy phosphates in the failing heart.
Increased fibroblast chymase production mediates procollagen autophagic digestion in volume overload.
Early detection of cardiac dysfunction in the type 1 diabetic heart using speckle-tracking based strain imaging.
Interleukin-10 inhibits chronic angiotensin II-induced pathological autophagy.
Phosphoinositide dependent protein kinase 1 is required for exercise-induced cardiac hypertrophy but not the associated mitochondrial adaptations.
Volume overload induces autophagic degradation of procollagen in cardiac fibroblasts.
Resolvin D1 activates the inflammation resolving response at splenic and ventricular site following myocardial infarction leading to improved ventricular function.
NEDDylation promotes endothelial dysfunction: A role for HDAC2.
P38α regulates SERCA2a function.
GLUT1 deficiency in cardiomyocytes does not accelerate the transition from compensated hypertrophy to heart failure.
Transmural heterogeneity of cellular level power output is reduced in human heart failure.
APpEaLINg therapeutic target for obesity cardiomyopathy?.
Cardiac immune cell remodeling after myocardial infarction.
Sequencing of mRNA identifies re-expression of fetal splice variants in cardiac hypertrophy.
Reduced expression of mitochondrial electron transport chain proteins from hibernating hearts relative to ischemic preconditioned hearts in the second window of protection.
Metabolic effects of glutamine on the heart: Anaplerosis versus the hexosamine biosynthetic pathway.
Regulation of myocardial metabolism by the cardiomyocyte circadian clock.
Genetic loss of insulin receptors worsens cardiac efficiency in diabetes.
Regional mechanics determine collagen fiber structure in healing myocardial infarcts.
Protein O-linked β-N-acetylglucosamine: A novel effector of cardiomyocyte metabolism and function.
Dynamic modulation of Ca2+ sparks by mitochondrial oscillations in isolated guinea pig cardiomyocytes under oxidative stress.
Expression of slow skeletal TnI in adult mouse hearts confers metabolic protection to ischemia.
Angiotensin-II type 1 receptor and NOX2 mediate TCF/LEF and CREB dependent WISP1 induction and cardiomyocyte hypertrophy.
A murine model of inducible, cardiac-specific deletion of STAT3: Its use to determine the role of STAT3 in the upregulation of cardioprotective proteins by ischemic preconditioning.
Cardiac cell therapy: Lessons from clinical trials.
Loss of interstitial collagen causes structural and functional alterations of cardiomyocyte subsarcolemmal mitochondria in acute volume overload.
Chronic AMD3100 antagonism of SDF-1α-CXCR4 exacerbates cardiac dysfunction and remodeling after myocardial infarction.
EMMPRIN activates multiple transcription factors in cardiomyocytes, and induces interleukin-18 expression via Rac1-dependent PI3K/Akt/IKK/NF-ΚB andMKK7/JNK/AP-1 signaling.
Contribution of extracellular matrix to the mechanical properties of the heart.
Corrigendum to "Anti-rat soluble IL-6 receptor antibody down-regulates cardiac IL-6 and improves cardiac function following trauma-hemorrhage" [J. Mol. Cell. Cardiol. 42 (2007) 620-630] (DOI:10.1016/j.yjmcc.2006.12.004).
Corrigendum to "Mechanism of IL-6-mediated cardiac dysfunction following trauma-hemorrhage" [J. Mol. Cell. Cardiol. 40 (2006) 570-579] (DOI:10.1016/j.yjmcc.2006.01.008).
Stretch-induced regulation of angiotensinogen gene expression in cardiac myocytes and fibroblasts: Opposing roles of JNK1/2 and p38α MAP kinases.
Skeletal myoblasts and cardiac repair.
Acrolein consumption exacerbates myocardial ischemic injury and blocks nitric oxide-induced PKCε signaling and cardioprotection.
Comparison of intracoronary and transendocardial delivery of allogeneic mesenchymal cells in a canine model of acute myocardial infarction.
PRDM6 is enriched in vascular precursors during development and inhibits endothelial cell proliferation, survival, and differentiation.
Stretch-induced MAP kinase activation in cardiac myocytes: Differential regulation through β1-integrin and focal adhesion kinase.
Erratum to "Cord blood and myocardial infarction: An uncertain wedding" [J. Mol. Cell. Cardiol. 42 (2007) 727-729] (DOI:10.1016/j.yjmcc.2007.02.006).
Regulation of cardiac substrate utilization by protein O-glycosylation.
Anti-rat soluble IL-6 receptor antibody down-regulates cardiac IL-6 and improves cardiac function following trauma-hemorrhage.
Cord blood and myocardial infarction: An uncertain wedding.
Glutamine-induced protection of isolated rat heart from ischemia/reperfusion injury is mediated via the hexosamine biosynthesis pathway and increased protein O-GlcNAc levels.
Enhanced ubiquitination of cytoskeletal proteins in pressure overloaded myocardium is accompanied by changes in specific E3 ligases.
Upregulation of mitochondrial respiratory complex IV by estrogen receptor-β is critical for inhibiting mitochondrial apoptotic signaling and restoring cardiac functions following trauma-hemorrhage.
A82. Bmx and c-Src mediates phosphorylation of p130Cas during cardiac hypertrophy.
Increased hexosamine biosynthesis and protein O-GlcNAc levels associated with myocardial protection against calcium paradox and ischemia.
Mechanism of IL-6-mediated cardiac dysfunction following trauma-hemorrhage.
Mechanism of cardioprotection following trauma-hemorrhagic shock by a selective estrogen receptor-β agonist: Up-regulation of cardiac heat shock factor-1 and heat shock proteins.
Prolonged oxidative stress inverts the cardiac force-frequency relation: Role of altered calcium handling and myofilament calcium responsiveness.
P66shc regulates endothelial NO production and endothelium-dependent vasorelaxation: Implications for age-associated vascular dysfunction.
Allopurinol and heart failure: A new use for an old drug?.
Post-infarction ventricular remodeling: An array of molecular events.
Diabetes and cardiac carbohydrate metabolism.
Diabetes, the hexosamine biosynthesis pathway and protein O-glycsoylation in the heart.
Increased carbohydrate oxidation and decreased fatty acid oxidation do not improve cardiac function after ischemia.
Oxidative and non-oxidative metabolic substrate fluxes in normal and ischemic rat heart.
Ischemic cardiac protection of high glucose/high insulin is not due to increased glucose oxidation.
Focal complex formation in adult cardiomyocytes is accompanied by the activation of β3 integrin and c-Src.
Beta-adrenergic receptor blockade modulates Bcl-X(S) expression and reduces apoptosis in failing myocardium..
β-Adrenergic receptor blockade modulates Bcl-XS expression and reduces apoptosis in failing myocardium.
A Novel TCTG(G/C) Direct Repeat and an A/T-Rich HMG2-Binding Site Control the Expression of the RatCardiacTroponin T Gene.
Alpha(1)-adrenoceptor subtypes mediate negative inotropy in myocardium from alpha(1A/C)-knockout and wild type mice..
08 Altered substrate provision and cardiac function during ischemia and reperfusion.
11 Type-2 diabetes leads to improved recovery of myocardial ischemia/reperfusion injury.
A comparison between NMR and GCMS 13C-isotopomer analysis in cardiac metabolism.
Cardiac carbohydrate metabolism in hearts from normal and type-2 diabetic rats.
The impact of type 2 diabetes on the regulation of cardiac energy metabolism and function.
Increased sensitivity of mitochondrial respiration to inhibition by nitric oxide in cardiac hypertrophy.
2-Tetradecylglycidic acid, an inhibitor of carnitine palmitoyltransferase-1, induces myocardial hypertrophy via the AT1 receptor.
Differential expression of angiotensin-converting enzyme and chymase in dogs with chronic mitral regurgitation.
Differential expression of natriuretic peptides and their receptors in volume overload cardiac hypertrophy in the rat.
Ischemic preconditioning increases iNOS transcript levels in conscious rabbits via a nitric oxide-dependent mechanism.
Nitric oxide effects on myocardial function and force-interval relations: Regulation of twitch duration.
Frequency-dependence of myocardial energetics in failing human myocardium as quantified by a new method for the measurement of oxygen consumption in muscle strip preparations.
Myocardial protection of contractile function after global ischemia by physiologic estrogen replacement in the ovariectomized rat.
The effects of hypertrophy and diabetes on cardiac pyruvate dehydrogenase activity.
Transforming growth factor-βs block cytokine induction of catalase and xanthine oxidase mRNA levels in cultured rat cardiac cells.
Autoinduction of mRNA and protein expression for transforming growth factor-βs in cultured cardiac cells.
Rapid conversion from β-MHC to α-MHC mRNA expression in embryonic rat ventricle cultured in oculo is not dependent on thyroid hormone or testosterone.
Sympathetic innervation modulates the expression of angiotensin II receptors in embryonic rat heart grafted in oculo.
Effect of temperature on Ca2+-dependent and mechanical modulators of relaxation in mammalian myocardium.
A 13C-NMR study of glucose oxidation in the intact functioning rat heart following diabetes-induced cardiomyopathy.
Changes in mitochondrial matrix free calcium in perfused rat hearts subjected to hypoxia-reoxygenation.
Effects of sympathetic innervation on size of myocytes in embryonic rat heart cultured in oculo.
Vagally induced depression of impulse propagation as a cause of atrial tachycardia.
Alterations in dietary sodium affect isoproterenol-induced cardiac hypertrophy.
Effects of repeated low calcium perfusion on the rat heart: A grandual induction of calcium related damage.
The relationship between cardiac function and metabolism in acute adriamycin-treated perfused rat hearts studied by 31P and 13C NMR spectroscopy.
Hypoxia-reoxygenation induced increase in cellular Ca2+ in myocytes and perfused hearts: the role of mitochondria.
Caronary vascular function and morphology in hydralazine treated DOCA salt rats.
Energy dependent insulin binding, internalization and degradation in isolated cardiac myocytes from normal and diabetic rats.
Xanthine oxidase as a source of free radical damage in myocardial ischemia.
Isolation and characterization of β1-adrenergic receptors from adult, rat cardiac myocytes.
Effect of peripheral sympathectomy on left ventricular ultrastructure in young spontaneously hypertensive rats..
Effect of peripheral sympathectomy on left ventricular ultrastructure in young spontaneously hypertensive rats.
Intramitochondrial inclusions in the myocardial cells of human hearts with coronary disease.
Myocardial hypertrophy and ventricular performance in the absence of hypertension in spontaneously hypertensive rats.
Preparation and characterization of plasma membrane of cardiac tissue.