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Chronobiological influence over cardiovascular function the good, the bad, and the ugly.
Race, Natriuretic Peptides, and High-Carbohydrate Challenge: A Clinical Trial.
Response by Pfeffer et al to Letter Regarding Article, "heart Failure with Preserved Ejection Fraction in Perspective".
Heart Failure with Preserved Ejection Fraction in Perspective.
Treatment of Resistant and Refractory Hypertension.
Convergences of Life Sciences and Engineering in Understanding and Treating Heart Failure.
Lower blood pressure thresholds raise the bar in pregnancy.
Nuclear focal adhesion kinase controls vascular smooth muscle cell proliferation and neointimal hyperplasia through GATA4-mediated cyclin D1 transcription.
Lack of Remuscularization Following Transplantation of Human Embryonic Stem Cell-Derived Cardiovascular Progenitor Cells in Infarcted Nonhuman Primates.
E2F1 suppresses oxidative metabolism and endothelial differentiation of bone marrow progenitor cells.
CCND2 Overexpression Enhances the Regenerative Potency of Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes: Remuscularization of Injured Ventricle.
Mitochondrial Reactive Oxygen Species in Lipotoxic Hearts Induce Post-Translational Modifications of AKAP121, DRP1, and OPA1 That Promote Mitochondrial Fission.
Can we engineer a human cardiac patch for therapy?.
Effective metabolic approaches for the energy starved failing heart: Bioenergetic resiliency via redundancy or something else?.
Global overview of the transnational alliance for regenerative therapies in cardiovascular syndromes (TACTICS) Recommendations: A comprehensive series of challenges and priorities of cardiovascular regenerative medicine.
Intra-cardiac release of extracellular vesicles shapes inflammation following myocardial infarction short communication.
Multifunctional role of chymase in acute and chronic tissue injury and remodeling.
Relationship between the efficacy of cardiac cell therapy and the inhibition of differentiation of human iPSC-Derived nonmyocyte cardiac cells into Myofibroblast-like cells.
Sprinting toward the optimal blood pressure target for hypertensive patients.
Transplanted mesenchymal stem cells reduce autophagic flux in infarcted hearts via the exosomal transfer of miR-125b.
PDK4 Inhibits Cardiac Pyruvate Oxidation in Late Pregnancy.
Meeting Report for the 2017 National Institutes of Health National Heart, Lung, and Blood Institute Progenitor Cell Biology Consortium: Cardiovascular Bioengineering Workshop and Symposium.
Immunomodulation is the key to cardiac repair.
Myocardial Tissue Engineering with Cells Derived from Human-Induced Pluripotent Stem Cells and a Native-Like, High-Resolution, 3-Dimensionally Printed Scaffold.
Elevating CXCR7 Improves Angiogenic Function of EPCs via Akt/GSK-3β/Fyn-Mediated Nrf2 Activation in Diabetic Limb Ischemia.
Reprogrammed Human Endothelial Cells.
From Microscale Devices to 3D Printing: Advances in Fabrication of 3D Cardiovascular Tissues.
A special report on the NHLBI initiative to study cellular and molecular mechanisms of arterial stiffness and its association with hypertension.
Lactate promotes synthetic phenotype in vascular smooth muscle cells.
Crossing into the Next Frontier of Cardiac Extracellular Matrix Research.
Meeting Report for NIH 2016 Progenitor Cell Biology Consortium Cardiovascular Tissue Engineering 2016.
Response by Zhou et al to Letter Regarding Article, Loss of Adult Cardiac Myocyte GSK-3 Leads to Mitotic Catastrophe Resulting in Fatal Dilated Cardiomyopathy.
The biological basis for cardiac repair after myocardial infarction.
Assessing Cardiac Metabolism.
Loss of adult cardiac myocyte GSK-3 leads to mitotic catastrophe resulting in fatal dilated cardiomyopathy.
Long-Term Outcome of Administration of c-kitPOS Cardiac Progenitor Cells after Acute Myocardial Infarction: Transplanted Cells Do not Become Cardiomyocytes, but Structural and Functional Improvement and Proliferation of Endogenous Cells Persist for at Least One Year.
A large-scale investigation of hypoxia-preconditioned allogeneic mesenchymal stem cells for myocardial repair in nonhuman primates: Paracrine activity without remuscularization.
CDKN2B Regulates TGFβ Signaling and Smooth Muscle Cell Investment of Hypoxic Neovessels..
The Nervous Heart.
Abstract 221: Chronic Alterations of the Tissue Dendritic Cell and Macrophage Network Underlies the Th1 Response in the Failing Heart.
Abstract 90: The Spleen Is an Essential Regulator of Systemic Neovascularization In Vivo.
Genetic Deficiency of Glutathione S-Transferase P Increases Myocardial Sensitivity to Ischemia-Reperfusion Injury.
Bach1 represses Wnt/β-catenin signaling and angiogenesis.
Embryonic Stem Cell-Derived Exosomes Promote Endogenous Repair Mechanisms and Enhance Cardiac Function Following Myocardial Infarction.
New Approaches in the Treatment of Hypertension.
The GSK-3 family as therapeutic target for myocardial diseases.
Abstract 60: The Role of Mononuclear Phagocytes in Pressure Overload Heart Failure.
PGC-1α induces SPP1 to activate macrophages and orchestrate functional angiogenesis in skeletal muscle.
OxLDL Triggers Retrograde Translocation of Arginase2 in Aortic Endothelial Cells via ROCK and Mitochondrial Processing Peptidase.
Sorafenib cardiotoxicity increases mortality after myocardial infarction.
Activation of AKT by O-linked N-Acetylglucosamine induces vascular calcification in diabetes mellitus.
It takes two to tango: Monocyte and macrophage duality in the infarcted heart.
Remodeling of the mononuclear phagocyte network underlies chronic inflammation and disease progression in heart failure critical importance of the cardiosplenic axis.
Patching the heart: Cardiac repair from within and outside.
Overexpression of endothelial nitric oxide synthase prevents diet-induced obesity and regulates adipocyte phenotype.
Metabolic remodeling in the hypertrophic heart: Fuel for thought.
Smooth muscle cell-specific runx2 deficiency inhibits vascular calcification.
Bioenergetic and functional consequences of cellular therapy: Activation of endogenous Cardiovascular progenitor cells.
Enhanced angiogenic and cardiomyocyte differentiation capacity of epigenetically reprogrammed mouse and human endothelial progenitor cells augments their efficacy for ischemic myocardial repair.
Orai1 determines calcium selectivity of an endogenous trpc heterotetramer channel.
Elucidating nature's solutions to heart, lung, and blood diseases and sleep disorders.
Inhalation of nitric oxide prevents ischemic brain damage in experimental stroke by selective dilatation of collateral arterioles.
Response to the letter by Edwards et al.
Animal models of heart failure a scientific statement from the American Heart Association.
Hydrogen sulfide as endothelium-derived hyperpolarizing factor sulfhydrates potassium channels.
Interleukin-10 deficiency impairs bone marrow-derived endothelial progenitor cell survival and function in ischemic myocardium.
PGC-1β deficiency accelerates the transition to heart failure in pressure overload hypertrophy.
Translational success stories: Angiotensin receptor 1 antagonists in heart failure.
Lack of primary cilia primes shear-induced endothelial-to-mesenchymal transition.
ATP production rate via creatine kinase or ATP synthase in Vivo: A novel superfast magnetization saturation transfer method.
Sustained hemodynamic stress disrupts normal circadian rhythms in calcineurin-dependent signaling and protein phosphorylation in the heart.
Genetic variation in NCAM1 contributes to left ventricular wall thickness in hypertensive families.
Late-breaking basic science abstracts from the American Heart Association's Scientific Sessions 2010.
PGC-1 coactivators in cardiac development and disease.
Increased endothelial exocytosis and generation of endothelin-1 contributes to constriction of aged arteries.
Preferential oxidation of triacylglyceride-derived fatty acids in heart is augmented by the nuclear receptor PPARα.
Decreased S-nitrosylation of tissue transglutaminase contributes to age-related increases in vascular stiffness.
Glycogen synthase kinase-3β regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo.
Peroxisome proliferator-activated receptor δ is an essential transcriptional regulator for mitochondrial protection and biogenesis in adult heart.
Career moves: Induced pluripotent cells from human aortic smooth muscle cells can efficiently redifferentiate into parental phenotype.
Nitro-fatty acid inhibition of neointima formation after endoluminal vessel injury.
Insulin-like growth factor binding protein-3 mediates vascular repair by enhancing nitric oxide generation.
Direct Relationship Between Protein O-GlcNAcylation and the Cardiomyocyte Circadian Clock.
Increased Cardiomyocyte 0-linked-beta-N-acetylglucosamine (0-GlcNAc) Levels Induced by Glucose Deprivation Mediated by Decreased 0-GlcNAcase Expression and Reduced Hexosamine Biosynthesis Flux.
O-linked-beta-N-acetylglucosamine (O-GlcNAc) Mediated Ischemic Cardioprotection Associated with Increased O-GlcNAcylation of Cytoskeletal Z-Line Proteins.
Inducible Expression of Active Protein Phosphatase-1 Inhibitor-1 Enhances Basal Cardiac Function and Protects Against Ischemia/Reperfusion Injury.
IL-10 inhibits inflammation and attenuates left ventricular remodeling after myocardial infarction via activation of STAT3 and suppression of HuR.
Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury.
Cell-free embryonic stem cell extract-mediated derivation of multipotent stem cells from NIH3T3 fibroblasts for functional and anatomical ischemic tissue repair.
Endothelial arginase II: A novel target for the treatment of atherosclerosis.
Negative-feedback loop attenuates hydrostatic lung edema via a cGMP-dependent regulation of transient receptor potential vanilloid 4.
Atrial natriuretic peptide inhibits transforming growth factor β-induced Smad signaling and myofibroblast transformation in mouse cardiac fibroblasts.
Role of mitochondrial dysfunction in insulin resistance.
Heme-Egr-1: New partners in atherosclerotic progression?.
Inducible NO synthase-dependent S-nitrosylation and activation of arginase1 contribute to age-related endothelial dysfunction.
Myocardial Smad4 is essential for cardiogenesis in mouse embryos.
Heme oxygenase-1 expression in macrophages plays a beneficial role in atherosclerosis.
Alternative splicing of ryanodine receptors modulates cardiomyocyte Ca2+ signaling and susceptibility to apoptosis.
Deoxymyoglobin is a nitrite reductase that generates nitric oxide and regulates mitochondrial respiration.
Myocyte nitroso-redox imbalance in sepsis: NO simple answer.
The role of O-GlcNAcylation in aging-related loss of estrogenic vasoprotection.
APOE4-VLDL inhibits the HDL-activated phosphatidylinositol 3-kinase/Akt pathway via the phosphoinositol phosphatase SHIP2.
Free radicals, mitochondria, and oxidized lipids: The emerging role in signal transduction in vascular cells.
Oxidized low-density lipoprotein-dependent endothelial arginase II activation contributes to impaired nitric oxide signaling.
Postinfarct cytokine therapy regenerates cardiac tissue and improves left ventricular function.
Coupled and uncoupled NOS: Separate but equal? Uncoupled NOS in endothelial cells is a critical pathway for intracellular signaling.
Cardiac myocyte apoptosis is associated with increased DNA damage and decreased survival in murine models of obesity.
Activation of the endothelial store-operated ISOC Ca 2+ channel requires interaction of protein 4.1 with TRPC4.
An apolipoprotein A-I mimetic works best in the presence of apolipoprotein A-I.
Oral small peptides render HDL antiinflammatory in mice and monkeys and reduce atherosclerosis in ApoE null mice.
Inhibition of lipopolysaccharide-induced inflammatory responses by an apolipoprotein AI mimetic peptide.
Glucosamine-induced cardioprotection mediated by the hexosamine biosynthesis pathway and increased levels of O-linked N-acetylglucosamine on nucleocytoplasmic proteins.
Essential role of a Ca2+-selective, store-operated current (ISOC) in endothelial cell permeability determinants of the vascular leak site.
Low-voltage-activated (T-type) calcium channels control proliferation of human pulmonary artery myocytes.
Increased PI3-kinase in presympathetic brain areas of the spontaneously hypertensive rat.
Cytokine-induced modulation of cardiac function.
Apurinic/apyrmidinic endonuclease 1 regulates endothelial NO production and vascular tone.
Antiinflammatory properties of HDL.
How do red blood cells dilate blood vessels?--Reply..
Effects of electrical shocks on Cai2+ and V m in myocyte cultures.
Nitric oxide protects against pathological ventricular remodeling: Reconsideration of the role of NO in the failing heart.
Upregulation of Alveolar Epithelial Active Na+ Transport Is Dependent on β2-Adrenergic Receptor Signaling.
Genome-Wide Scan Identifies Novel QTLs for Cholesterol and LDL Levels in F2[Dahl RxS] Intercross Rats.
Nonlinear Changes of Transmembrane Potential, during Electrical Shocks: Role of Membrane Electroporation.
CaV3.1 (α1G) T-type Ca2+ channels mediate vaso-occlusion of sickled erythrocytes in lung microcirculation.
Nitric oxide regulation of myocardial contractility and calcium cycling: Independent impact of neuronal and endothelial nitric oxide synthases.
Gene therapy with inducible nitric oxide synthase protects against myocardial infarction via a cyclooxygenase-2-dependent mechanism.
Sustained reentry in the left ventricle of fibrillating pig hearts.
Vectorial sodium transport across the mammalian alveolar epithelium: It occurs but through which cells?.
Regional differences in ventricular fibrillation in the open-chest porcine left ventricle.
Effects of S-nitrosation and cross-linking of hemoglobin on hypoxic pulmonary vasoconstriction in isolated rat lungs.
Gene dosage-dependent effects of cardiac-specific overexpression of the A3 adenosine receptor.
Optical mapping of arrhythmias induced by strong electrical shocks in myocyte cultures.
Recent fibrillation studies: Attempts to wrest order from disorder.
Nitric oxide synthase (NOS2) mutation in Dahl/Rapp rats decreases enzyme stability.
Autonomous and growth factor - Induced hypertrophy in cultured neonatal mouse cardiac myocytes: Comparison with rat.
Nonlinear changes of transmembrane potential during defibrillation shocks: Role of Ca2+ current.
Hydrogen peroxide- and peroxynitrite-induced mitochondrial DNA damage and dysfunction in vascular endothelial and smooth muscle cells.
Fibrillating myocardium: Rabbit warren or beehive?.
Elements in the Cardiac Troponin T Gene Conferring Specific Expression in Cardiac Muscle of Transgenic Mice.
The angiotensin II-dependent association of Jak2 and c-Src requires the N-terminus of Jak2 and the SH2 domain of c-Src.
Chemiluminescent detection of oxidants in vascular tissue: Lucigenin but not coelenterazine enhances superoxide formation.
Incidence, evolution, and spatial distribution of functional reentry during ventricular fibrillation in pigs.
Spatial changes in the transmembrane potential during extracellular electric stimulation.
Importance of E-selectin for firm leukocyte adhesion in vivo.
ATP-sensitive K+ channels, adenosine, and nitric oxide-mediated mechanisms account for coronary vasodilation during exercise.
Activation of cardiac tissue by extracellular electrical shocks: Formation of 'secondary sources' at intercellular clefts in monolayers of cultured myocytes.
Cyclic GMP-dependent protein kinase inhibits osteopontin and thrombospondin production in rat aortic smooth muscle cells.
Cross talk between angiotensin AT1 and α1-adrenergic receptors: Angiotensin II downregulates α(1a)-adrenergic receptor subtype mRNA and density in neonatal rat cardiac myocytes.
Anisotropic Activation Spread in Heart Cell Monolayers Assessed by High-Resolution Optical Mapping: Role of Tissue Discontinuities.
Functional and structural assessment of intercellular communication: Increased conduction velocity and enhanced connexin expression in dibutyryl cAMP-treated cultured cardiac myocytes.
Spatial changes in transmembrane potential during extracellular electrical shocks in cultured monolayers of neonatal rat ventricular myocytes.
23Na and 31P nuclear magnetic resonance studies of ischemia-induced ventricular fibrillation. Alterations of intracellular Na+ and cellular energy..
Formation of F2-isoprostanes during oxidation of human low-density lipoprotein and plasma by peroxynitrite.
Altered pulmonary microvascular reactivity to norepinephrine in canine pacing-induced heart failure..
Anisotropic conduction in monolayers of neonatal rat heart cells cultured on collagen substrate.
Cloning of the rat α(1C)-adrenergic receptor from cardiac myocytes: α(1C), α(1B), and α(1D) mRNAs are present in cardiac myocytes but not in cardiac fibroblasts.
Computer simulations of three-dimensional propagation in ventricular myocardium: Effects of intramural fiber rotation and inhomogeneous conductivity on epicardial activation.
Coronary pressure-flow relation in left ventricular hypertrophy: Importance of changes in back pressure versus changes in minimum resistance.
Microscopic conduction in cultured strands of neonatal rat heart cells measured with voltage-sensitive dyes.
Kinetics of restitution of left ventricular relaxation.
Heterogeneity within the ventricular wall. Electrophysiology and pharmacology of epicardial, endocardial, and M cells.
Correlation between transmural high energy phosphate levels and myocardial blood flow in the presence of graded coronary stenosis.
Differences in myosin isoform expression in the subepicardial and subendocardial myocardium during cardiac hypertrophy in the rat.
Differences in the electrophysiological response to canine ventricular subendocardium and subepicardium to acetylcholine and isoproterenol. A direct effect of acetylcholine in ventricular myocardium.
Increased ischemic injury but decreased hypoxic injury in hypertrophied rat hearts.
Morphological development of the rat heart growing in oculo in the absence of hemodynamic work load.
Vagally induced block and delayed conduction as a mechanism for circus movement tachycardia in frog atria.
Vascular remodeling and improvement of coronary reserve after hydralazine treatment in spontaneously hypertensive rats.
Transient outward current prominent in canine ventricular epicardium but not endocardium.
Sympathetic innervation alters growth and intrinsic heart rate of fetal rat atria maturing in oculo.
Histamine H1 receptor antagonists inhibit autoregulation of renal blood flow in the dog.
Effect of acute hypoxia on the pulmonary conversion of angiotensin I to angiotensin II in dogs.
Autoregulatory responses of superficial nephrons and their association with sodium excretion during arterial pressure alterations in the dog.
Development of left ventricular hypertrophy in young spontaneously hypertensive rats after peripheral sympathectomy.
Relation of renal hemodynamics to metabolism of angiotensin II by the canine kidney.
Structural requirements for substrates and inhibitors of angiotensin I converting enzyme in vivo and in vitro.
Mechanism of renal handling of angiotensin II in the dog.
Substrate requirements for angiotensin I conversion in vivo and in vitro..
Mechanism of pulmonary conversion of angiotensin I to angiotensin II in the dog..
In-vivo and in-vitro conversion of angiotensin I to angiotensin II in dog blood..