| Year |
Title |
Altmetric |
| 2021
|
Repurposing Nintedanib for pathological cardiac remodeling and dysfunction.
Pharmacological Research.
169.
2021
|
|
| 2021
|
Tom Force (1951–2020): Our Dearest Friend, Our Mentor, and a Brilliant Cardiovascular Scientist
2021
|
|
| 2021
|
Targeting 5-HT2BReceptor Signaling Prevents Border Zone Expansion and Improves Microstructural Remodeling after Myocardial Infarction.
Circulation.
1317-1330.
2021
|
|
| 2020
|
The BDNF rs6265 polymorphism is a modifier of cardiomyocyte contractility and dilated cardiomyopathy.
International Journal of Molecular Sciences.
21:1-16.
2020
|
|
| 2020
|
Cardiotoxicity of the BCR-ABL1 tyrosine kinase inhibitors: Emphasis on ponatinib.
International Journal of Cardiology.
316:214-221.
2020
|
|
| 2020
|
Mouse Models of Heart Failure with Preserved or Reduced Ejection Fraction.
American Journal of Pathology.
190:1596-1608.
2020
|
|
| 2020
|
A Pharmacovigilance Study of Hydroxychloroquine Cardiac Safety Profile: Potential Implication in COVID-19 Mitigation..
Journal of Clinical Medicine.
9.
2020
|
|
| 2020
|
Deletion of Cardiomyocyte Glycogen Synthase Kinase-3 Beta (GSK-3β) Improves Systemic Glucose Tolerance with Maintained Heart Function in Established Obesity.
Cells.
9.
2020
|
|
| 2020
|
Neutrophil-derived S100A8/A9 amplify granulopoiesis after myocardial infarction.
Circulation.
1080-1094.
2020
|
|
| 2019
|
Cardiomyocyte Homeodomain-Interacting Protein Kinase 2 Maintains Basal Cardiac Function via Extracellular Signal-Regulated Kinase Signaling.
Circulation.
140:1820-1833.
2019
|
|
| 2019
|
Cadherin-11 blockade reduces inflammation-driven fibrotic remodeling and improves outcomes after myocardial infarction.
JCI insight.
4.
2019
|
|
| 2019
|
Cardiomyocyte-GSK-3α promotes mPTP opening and heart failure in mice with chronic pressure overload.
Journal of Molecular and Cellular Cardiology.
130:65-75.
2019
|
|
| 2019
|
Ponatinib-induced cardiotoxicity: Delineating the signalling mechanisms and potential rescue strategies.
Cardiovascular Research.
115:966-977.
2019
|
|
| 2019
|
Cardiomyocyte SMAD4-Dependent TGF-β Signaling is Essential to Maintain Adult Heart Homeostasis.
JACC: Basic to Translational Science.
4:41-53.
2019
|
|
| 2019
|
Generation of Nppa-tagBFP reporter knock-in mouse line for studying cardiac chamber specification
2019
|
|
| 2019
|
IL-10–producing B cells are enriched in murine pericardial adipose tissues and ameliorate the outcome of acute myocardial infarction
2019
|
|
| 2019
|
Inhibition of GSK-3 to induce cardiomyocyte proliferation: a recipe for in situ cardiac regeneration.
Cardiovascular Research.
115:20-30.
2019
|
|
| 2018
|
Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model.
International Journal of Cardiology.
259:145-152.
2018
|
|
| 2017
|
Chronic Neuregulin-1β Treatment Mitigates the Progression of Postmyocardial Infarction Heart Failure in the Setting of Type 1 Diabetes Mellitus by Suppressing Myocardial Apoptosis, Fibrosis, and Key Oxidant-Producing Enzymes.
Journal of Cardiac Failure.
23:887-899.
2017
|
|
| 2017
|
Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis.
Journal of Molecular and Cellular Cardiology.
110:109-120.
2017
|
|
| 2017
|
Mechanistic Insights of Empagliflozin-Mediated Cardiac Benefits: Nearing the Starting Line: Editorial to: “Empagliflozin Improves Left Ventricular Diastolic Dysfunction in a Genetic Model of Type 2 Diabetes” by N. Hammoudi et al..
Cardiovascular Drugs and Therapy.
31:229-232.
2017
|
|
| 2017
|
Activation of the amino acid response pathway blunts the effects of cardiac stress
2017
|
|
| 2016
|
Response by Zhou et al to Letter Regarding Article, Loss of Adult Cardiac Myocyte GSK-3 Leads to Mitotic Catastrophe Resulting in Fatal Dilated Cardiomyopathy.
Circulation Research.
119:e29-e30.
2016
|
|
| 2016
|
Loss of adult cardiac myocyte GSK-3 leads to mitotic catastrophe resulting in fatal dilated cardiomyopathy.
Circulation Research.
118:1208-1222.
2016
|
|
| 2015
|
The GSK-3 family as therapeutic target for myocardial diseases.
Circulation Research.
116:138-149.
2015
|
|
| 2014
|
Cardiomyocyte-specific deletion of Gsk3α mitigates post-myocardial infarction remodeling, contractile dysfunction, and heart failure.
Journal of the American College of Cardiology.
64:696-706.
2014
|
|
| 2014
|
Cardiac fibroblast glycogen synthase kinase-3β regulates ventricular remodeling and dysfunction in ischemic heart.
Circulation.
130:419-430.
2014
|
|
| 2014
|
Sorafenib cardiotoxicity increases mortality after myocardial infarction.
Circulation Research.
114:1700-1712.
2014
|
|
| 2014
|
Prevention of liver cancer cachexia-induced cardiac wasting and heart failure.
European Heart Journal.
35:932-941.
2014
|
|
| 2014
|
Troponin I-interacting protein kinase - A novel cardiac-specific kinase, emerging as a molecular target for the treatment of cardiac disease.
Circulation Journal.
78:1514-1519.
2014
|
|
| 2013
|
Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network.
International Journal of Cardiology.
168:3884-3895.
2013
|
|
| 2013
|
Caveolin and β1-integrin coordinate angiotensinogen expression in cardiac myocytes.
International Journal of Cardiology.
168:436-445.
2013
|
|
| 2013
|
GSK-3α is a central regulator of age-related pathologies in mice.
Journal of Clinical Investigation.
123:1821-1832.
2013
|
|
| 2013
|
Cancer genetics and the cardiotoxicity of the therapeutics.
Journal of the American College of Cardiology.
61:267-274.
2013
|
|
| 2012
|
Glycogen synthase kinase-3α limits ischemic injury, cardiac rupture, post-myocardial infarction remodeling and death.
Circulation.
125:65-75.
2012
|
|
| 2011
|
Rac1 and RhoA differentially regulate angiotensinogen gene expression in stretched cardiac fibroblasts.
Cardiovascular Research.
90:88-96.
2011
|
|
| 2011
|
A novel cardioprotective p38-MAPK/mTOR pathway.
Experimental Cell Research.
317:2938-2949.
2011
|
|
| 2010
|
GSK-3α directly regulates β-adrenergic signaling and the response of the heart to hemodynamic stress in mice.
Journal of Clinical Investigation.
120:2280-2291.
2010
|
|
| 2010
|
Glycogen synthase kinase-3β regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo.
Circulation Research.
106:1635-1645.
2010
|
|
| 2009
|
Anthrax toxin: Pathologic effects on the cardiovascular system.
Frontiers in Bioscience.
14:2335-2357.
2009
|
|
| 2009
|
Integrins and proximal signaling mechanisms in cardiovascular disease.
Frontiers in Bioscience.
14:2307-2334.
2009
|
|
| 2008
|
Stretch-induced regulation of angiotensinogen gene expression in cardiac myocytes and fibroblasts: Opposing roles of JNK1/2 and p38α MAP kinases.
Journal of Molecular and Cellular Cardiology.
45:770-778.
2008
|
|
| 2007
|
Stretch-induced MAP kinase activation in cardiac myocytes: Differential regulation through β1-integrin and focal adhesion kinase.
Journal of Molecular and Cellular Cardiology.
43:137-147.
2007
|
|
| 2007
|
Lethal and edema toxins of anthrax induce distinct hemodynamic dysfunction.
Frontiers in Bioscience.
12:4670-4675.
2007
|
|
| 2007
|
Integrins: Novel therapeutic targets for cardiovascular diseases.
Cardiovascular and Hematological Agents in Medicinal Chemistry.
5:109-132.
2007
|
|
| 2000
|
Stress induced phosphate solubilization in bacteria isolated from alkaline soils.
FEMS Microbiology Letters.
182:291-296.
2000
|
|
