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Hind Lal
Associate Professor
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E-mail
hindlal@uabmc.edu
hindlal@uab.edu
Phone
205-996-4219
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Hind Lal
Associate Professor
Positions
Scientist (C)
,
Comprehensive Cardiovascular Center
,
School of Medicine
2019 -
Associate Professor (P)
,
Medicine - Cardiovascular Disease
,
Department of Medicine
2019 -
Publications
Research
Background
Contact
Publications
Selected Publications
Academic Article
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Year
Title
Altmetric
2021
Targeting 5-HT2B Receptor Signaling Prevents Border Zone Expansion and Improves Microstructural Remodeling After Myocardial Infarction.
.
Circulation
. 143:1317-1330.
2021
2020
The BDNF rs6265 polymorphism is a modifier of cardiomyocyte contractility and dilated cardiomyopathy
.
International Journal of Molecular Sciences
. 21:1-16.
2020
2020
Cardiotoxicity of the BCR-ABL1 tyrosine kinase inhibitors: Emphasis on ponatinib
.
International Journal of Cardiology
. 316:214-221.
2020
2020
Mouse Models of Heart Failure with Preserved or Reduced Ejection Fraction
.
American Journal of Pathology
. 190:1596-1608.
2020
2020
A Pharmacovigilance Study of Hydroxychloroquine Cardiac Safety Profile: Potential Implication in COVID-19 Mitigation.
.
Journal of Clinical Medicine
. 9.
2020
2020
Deletion of Cardiomyocyte Glycogen Synthase Kinase-3 Beta (GSK-3β) Improves Systemic Glucose Tolerance with Maintained Heart Function in Established Obesity
.
Cells
. 9.
2020
2020
Neutrophil-derived S100A8/A9 amplify granulopoiesis after myocardial infarction
.
Circulation
. 1080-1094.
2020
2019
Cardiomyocyte Homeodomain-Interacting Protein Kinase 2 Maintains Basal Cardiac Function via Extracellular Signal-Regulated Kinase Signaling
.
Circulation
. 140:1820-1833.
2019
2019
Cadherin-11 blockade reduces inflammation-driven fibrotic remodeling and improves outcomes after myocardial infarction
.
JCI insight
. 4.
2019
2019
Cardiomyocyte-GSK-3α promotes mPTP opening and heart failure in mice with chronic pressure overload
.
Journal of Molecular and Cellular Cardiology
. 130:65-75.
2019
2019
Ponatinib-induced cardiotoxicity: Delineating the signalling mechanisms and potential rescue strategies
.
Cardiovascular Research
. 115:966-977.
2019
2019
Cardiomyocyte SMAD4-Dependent TGF-β Signaling is Essential to Maintain Adult Heart Homeostasis
.
JACC: Basic to Translational Science
. 4:41-53.
2019
2019
Generation of Nppa-tagBFP reporter knock-in mouse line for studying cardiac chamber specification
2019
2019
IL-10–producing B cells are enriched in murine pericardial adipose tissues and ameliorate the outcome of acute myocardial infarction
2019
2019
Inhibition of GSK-3 to induce cardiomyocyte proliferation: a recipe for in situ cardiac regeneration
.
Cardiovascular Research
. 115:20-30.
2019
2018
Cardiomyocyte-specific deletion of GSK-3β leads to cardiac dysfunction in a diet induced obesity model
.
International Journal of Cardiology
. 259:145-152.
2018
2017
Chronic Neuregulin-1β Treatment Mitigates the Progression of Postmyocardial Infarction Heart Failure in the Setting of Type 1 Diabetes Mellitus by Suppressing Myocardial Apoptosis, Fibrosis, and Key Oxidant-Producing Enzymes
.
Journal of Cardiac Failure
. 23:887-899.
2017
2017
Entanglement of GSK-3β, β-catenin and TGF-β1 signaling network to regulate myocardial fibrosis
.
Journal of Molecular and Cellular Cardiology
. 110:109-120.
2017
2017
Mechanistic Insights of Empagliflozin-Mediated Cardiac Benefits: Nearing the Starting Line: Editorial to: “Empagliflozin Improves Left Ventricular Diastolic Dysfunction in a Genetic Model of Type 2 Diabetes” by N. Hammoudi et al.
.
Cardiovascular Drugs and Therapy
. 31:229-232.
2017
2017
Activation of the amino acid response pathway blunts the effects of cardiac stress
2017
2016
Response by Zhou et al to Letter Regarding Article, Loss of Adult Cardiac Myocyte GSK-3 Leads to Mitotic Catastrophe Resulting in Fatal Dilated Cardiomyopathy
.
Circulation Research
. 119:e29-e30.
2016
2016
Loss of adult cardiac myocyte GSK-3 leads to mitotic catastrophe resulting in fatal dilated cardiomyopathy
.
Circulation Research
. 118:1208-1222.
2016
2015
The GSK-3 family as therapeutic target for myocardial diseases
.
Circulation Research
. 116:138-149.
2015
2014
Cardiomyocyte-specific deletion of Gsk3α mitigates post-myocardial infarction remodeling, contractile dysfunction, and heart failure
.
Journal of the American College of Cardiology
. 64:696-706.
2014
2014
Cardiac fibroblast glycogen synthase kinase-3β regulates ventricular remodeling and dysfunction in ischemic heart
.
Circulation
. 130:419-430.
2014
2014
Sorafenib cardiotoxicity increases mortality after myocardial infarction
.
Circulation Research
. 114:1700-1712.
2014
2014
Prevention of liver cancer cachexia-induced cardiac wasting and heart failure
.
European Heart Journal
. 35:932-941.
2014
2014
Troponin I-interacting protein kinase - A novel cardiac-specific kinase, emerging as a molecular target for the treatment of cardiac disease
.
Circulation Journal
. 78:1514-1519.
2014
2013
Anthrax lethal toxin induces acute diastolic dysfunction in rats through disruption of the phospholamban signaling network
.
International Journal of Cardiology
. 168:3884-3895.
2013
2013
Caveolin and β1-integrin coordinate angiotensinogen expression in cardiac myocytes
.
International Journal of Cardiology
. 168:436-445.
2013
2013
GSK-3α is a central regulator of age-related pathologies in mice
.
Journal of Clinical Investigation
. 123:1821-1832.
2013
2013
Cancer genetics and the cardiotoxicity of the therapeutics
.
Journal of the American College of Cardiology
. 61:267-274.
2013
2012
Glycogen synthase kinase-3α limits ischemic injury, cardiac rupture, post-myocardial infarction remodeling and death
.
Circulation
. 125:65-75.
2012
2011
Rac1 and RhoA differentially regulate angiotensinogen gene expression in stretched cardiac fibroblasts
.
Cardiovascular Research
. 90:88-96.
2011
2011
A novel cardioprotective p38-MAPK/mTOR pathway
.
Experimental Cell Research
. 317:2938-2949.
2011
2010
GSK-3α directly regulates β-adrenergic signaling and the response of the heart to hemodynamic stress in mice
.
Journal of Clinical Investigation
. 120:2280-2291.
2010
2010
Glycogen synthase kinase-3β regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo
.
Circulation Research
. 106:1635-1645.
2010
2009
Anthrax toxin: Pathologic effects on the cardiovascular system
.
Frontiers in Bioscience
. 14:2335-2357.
2009
2009
Integrins and proximal signaling mechanisms in cardiovascular disease
.
Frontiers in Bioscience
. 14:2307-2334.
2009
2008
Stretch-induced regulation of angiotensinogen gene expression in cardiac myocytes and fibroblasts: Opposing roles of JNK1/2 and p38α MAP kinases
.
Journal of Molecular and Cellular Cardiology
. 45:770-778.
2008
2007
Stretch-induced MAP kinase activation in cardiac myocytes: Differential regulation through β
1
-integrin and focal adhesion kinase
.
Journal of Molecular and Cellular Cardiology
. 43:137-147.
2007
2007
Lethal and edema toxins of anthrax induce distinct hemodynamic dysfunction
.
Frontiers in Bioscience
. 12:4670-4675.
2007
2007
Integrins: Novel therapeutic targets for cardiovascular diseases
.
Cardiovascular and Hematological Agents in Medicinal Chemistry
. 5:109-132.
2007
2000
Stress induced phosphate solubilization in bacteria isolated from alkaline soils
.
FEMS Microbiology Letters
. 182:291-296.
2000
Chapter
Filter
Year
Title
Altmetric
2009
Molecular Signaling Mechanisms of Myocardial Stretch: Implications for Heart Disease
2009
Research
Principal Investigator On
Novel Mechanisms of Cardiac Function and Dysfunction
awarded by
National Heart, Lung, and Blood Institute/NIH/DHHS
2019 - 2023
Signaling Mechanisms Governing Myocardial Fibrosis in Diseased Heart
awarded by
National Heart, Lung, and Blood Institute/NIH/DHHS
2020 - 2022
Role of GSK-3-alpha in the Regulation of Myocardial Fibrosis
awarded by
American Heart Association
2019 - 2020
Background
Education And Training
Doctor of Philosophy in Microbiology,
2006
Contact
Full Name
Hind
Lal